Berberine: A dual anti-HIV and anti- cervical cancer compound.
We report the effects of berberine (BBR), a benzylisoquinoline alkaloid small molecule on inhibition of HIV infection of cervical cancer cells.
Cervical Cancer · LIVING RESEARCH LIBRARY
Ongoing Research for Cervical Cancer Using Advanced Alternative Medicine
Explore positive published findings by substance, cancer biology, treatment interaction and quality-of-life outcome.
Every entry includes the original study title, year, concise finding and a direct PubMed link.
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Research organized by substance and finding
Browse 17 source-linked studies with the full title, publication year, research focus and a concise finding. Select any linked substance to move directly to its corresponding Sunridge page.
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Curcumin Nanoformulation for Cervical Cancer Treatment.
Curcumin (CUR) is a well-known natural compound, which exhibits excellent anti-cancer potential by regulating many proliferative, oncogenic, and chemo-resistance associated genes/proteins.
EGCG remodels the TGF-β cervical cancer micro-environment towards immune responsiveness.
EGCG treatment under TGF-β, reduced PD-L1, CD55 and CD46 expression, decreased viability, metabolic activity and MMP-2 secretion, while inducing apoptosis in SiHa co-culture.
Ginseng Glucosyl Oleanolate Suppresses Cervical Cancer via Simultaneous Inhibition of Glycolysis and Oxidative Phosphorylation In Vivo/Vitro.
Ginseng, a popular functional food, owes its benefits mainly to ginsenosides with reported antitumor activity that have been widely reported for its antitumor activity.
Identification of Glutathione Synthetase as a Therapeutic Target for Cervical Cancer via Combining Bioinformatics and Experimental Validation.
Drug sensitivity analysis linked GSS to vorinostat, which inhibits tumour growth by suppressing the PI3K/Akt pathway and downregulating GSS.
Retraction Note: Melatonin enhances TNF-α-mediated cervical cancer HeLa cells death via suppressing CaMKII/Parkin/mitophagy axis.
The study reports retraction Note: Melatonin enhances TNF-α-mediated cervical cancer HeLa cells death via suppressing CaMKII/Parkin/mitophagy axis.
Characterization of cervical microbiota in cervical intraepithelial neoplasia and cervical cancer using low-coverage whole genome sequencing.
CC microbiota showed significant Bacteroidetes enrichment (82% at class level) and reduced Firmicutes abundance.
Characterization of the vaginal microbiome of postmenopausal patients receiving chemoradiation for locally advanced cervical cancer.
We hypothesized that characteristics of the microbiome might correlate with treatment response.
The intratumoral microbiota heterogenicity is related to the prognosis and tumorigenesis of cervical cancer.
The microbiome can influence cancer development by modulating inflammation, immune responses and metabolic pathways.
The Role of Intratumor Microbiomes in Cervical Cancer Metastasis.
The relationship between intratumor microbiomes and cervical cancer metastasis, however, remains unclear.
Zhidai Decoction Inhibits Cervical Cancer through Regulation of Vaginal Microbiota.
In conclusion, ZDD displayed inhibitory effect on cervical cancer, and it might be based on restoring the balance of vaginal microbiota.
Investigating the potential mechanism of quercetin against cervical cancer.
Cervical cancer is emerging as a potential target of increased susceptibility to coronavirus disease-2019 (COVID-19), leading to compromised survival rates.
Quercetin up-regulates the expression of tumor-suppressive microRNAs in human cervical cancer.
The results from this study show that quercetin has the potential to prevent cervical cancer by regulating the expression of tumor-suppressive miRNAs.
Resveratrol and Cervical Cancer: A New Therapeutic Option.
Among the herb-based components, resveratrol has several beneficial effects given its anticancer activities (e.g., anti-angiogenesis, anti-proliferation, anti-metastatic and pro-apoptotic).
Sodium selenite inhibits cervical cancer growth via ROS mediated AMPK/FOXO3a /GADD45a axis.
HeLa cell xenografts in female nude mice showed tumor growth retardation, with no obvious liver and kidney toxicity, after being intraperitoneally injected with 3 mg/kg sodium selenite (SS) for 14 days.
Sodium selenite inhibits cervical cancer progression via ROS-mediated suppression of glucose metabolic reprogramming.
Sodium selenite (SS) treated HeLa and SiHa cells were assessed for proliferation using the CCK-8 assay and immunofluorescence.
Cholecalciferol induces apoptosis via autocrine metabolism in epidermoid cervical cancer cells.
The authors conclude that autocrine activation of cholecalciferol to calcidiol may mediate VDR signalling of growth inhibition and apoptosis in SiHa cells.
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