A migraine headache is accompanied by or preceded by warnings including tingling in extremities, nausea, blind spots, light flashes and sensitivity to light and sound is a migraine headache. The pain associated with a headache is often debilitating because of its severity which may last a few hours to several days.
Approximately twelve percent of the population suffers from migraine headaches. Symptoms of Migraine Headache
Symptoms of a migraine can occur sometime before a headache, immediately before, during and after a headache. All migraines are not the same, some symptoms include:
- Moderate to severe pain head pain, usually confined to one side.
- Physical activity typically increases the pain.
- Throbbing Head Pain
- Increased sensitivity to light and sound.
- Nausea, stomach upset, diarrhea.
- Sweating, chills, and sensitivity to temperature.
Chronic Migraines
Chronic migraine headaches are those headaches where a patient experiences symptoms for half the month over a period of six months. Headaches are a leading causes of emergency room visits, particularly among women during their reproductive years. However, men also suffer from migraine headaches.
Triggers of Migraine Headache
- Physical stress such as from a hard workout.
- Emotional stress, tension, or excitement.
- Allergies and allergic reactions.
- Strong smells and certain perfumes.
- Flickering or bright lights.
- Skipping meals, juice cleanses or fasting. tion and urinary retention
- Hormonal changes including menstrual cycle.
- Not drinking enough water, being dehydrated.
- Drinking alcohol including red wine.
- Eating food with nitrates and other foods.
Alternative Treatment for Migraine Headaches
Alternative treatment for migraine headaches is offered at Sunridge Medical and includes first discovering the root cause of headaches and treating. We create a custom designed treatment plan to treat the whole body and not just the symptoms of migraine headaches and bring relief once and for all.
References on Migraine Headaches
Bulboacă, A. E., Bolboacă, S. D., Stănescu, I. C., Sfrângeu, C. A., & Bulboacă, A. C. (2017). Preemptive Analgesic and Antioxidative Effect of Curcumin for Experimental Migraine. BioMed research international, 2017, 4754701. https://doi.org/10.1155/2017/4754701
Objective Our study aimed to investigate the analgesic and antioxidative stress effects of Curcumin (CC) in experimental migraine induced by Nitroglycerin (NTG) on rats, compared with Indomethacin (ID) and Propranolol (PP) treatments.
Material and Methods Five groups of 10 rats treated i.p. were investigated: control group (healthy rats) injected with saline solution (0.9%), NTG-control group injected with NTG (1 mg/100 gbw, bw = body weight), and three groups with pretreatment applied 30 min.
Previous to the formalin test (NTG + CC group: Curcumin (10 mg/100 gbw), NTG + PP group: Propranolol (100 μg/100 gbw), and NTG + ID group: Indomethacin (0.5 mg/100 gbw)).
Formalin test was performed and number of flinches and shakes were counted. Several oxidative stress parameters were also assessed.
Results The smallest values of malondialdehyde (MDA), nitric oxide (NOx), and total oxidative status (TOS) were observed on NTG + CC with significant differences as compared with the control group (p < 0.0001). The group pretreated with Curcumin proved significantly smaller number of flinches and shakes compared with both NTG + PP and NTG + ID.
Conclusion Our study demonstrates a superior activity of Curcumin not only versus control, but also versus Propranolol and Indomethacin.
Haggiag S, Prosperini L, Stasolla A, Gerace C, Tortorella C, Gasperini C. Ozone-induced encephalopathy: A novel iatrogenic entity. Eur J Neurol. 2021 Aug;28(8):2471-2478. doi: 10.1111/ene.14793. Epub 2021 Mar 19. PMID: 33657263. https://pubmed.ncbi.nlm.nih.gov/33657263/
Background and purpose: Ozone-based treatments can be associated with central nervous system toxicity, which we have termed ozone-induced encephalopathy (OIE). A detailed description of its phenotype is lacking.
Methods: Three cases with findings suggestive of OIE are presented, and the literature is reviewed.
Results: Case 1 is a healthy 59-year-old man presenting with loss of consciousness, cortical blindness, restlessness, and anterograde amnesia immediately following a cervical ozone-therapy (OT) session for chronic neck pain. Brain magnetic resonance imaging (MRI) on admission was normal.
A follow-up scan demonstrated a subtle increased T2 fluid-attenuated inversion recovery signal within the left cerebellum; an echocardiography showed a patent foramen ovale (PFO).
Case 2 is a 56-year-old woman with history of migraine, PFO, and lumbar pain who presented with headache, bilateral visual impairment, motor dysphasia, and agitation. All her symptoms began immediately after lumbar OT. Her brain MRI was negative. Case 3 is a healthy 27-year-old man who complained of vertigo and mild blurred vision 5 min following a cervical ozone injection.
His neurological examination and brain MRI were normal. All three patients had full recovery within 48 h. We found eight additional cases of OIE in the literature.
Conclusions: OIE should be considered in patients presenting with neurological symptoms in close relation to OT. OIE is likely a novel iatrogenic entity with a complex pathogenesis; it is probably underreported because it mimics other neurological conditions.
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