Multiple Sclerosis Treatment Options
At Sunridge Medical we stand at the forefront of alternative treatments for patients with multiple sclerosis and other neurologic conditions using the experience and expertise of our medical team. We specialize in comprehensive testing, and individualized treatment programs prepared for each of our patients to address and care for multiple sclerosis. There is no single known cause of MS, yet there are a number of contributing factors can lead to the onset and progression of the disease including genetics, vitamin D status, infections including Lyme disease, and heavy metal exposure and retention.
Sunridge treats relapsing-remitting multiple sclerosis, its accompanying symptoms and progression. Contacting our patient care representatives will begin your journey towards healing.
Revolutionizing Treatment of Multiple Sclerosis
- Genetics always play a role in health and disease and all autoimmune illnesses have a genetic component, yet 85% of multiple sclerosis patients do not have an affected relative and only 30% of identical twins develop multiple sclerosis if the other twin already has it.
- Clinical studies have pointed out that vitamin D deficiency may influence the development and progression of MS.
- Evidence shows there is an infectious component, etiology or contribution in the majority of MS cases. Infections that have a possible role in the pathogenesis of MS and perhaps other neurodegenerative diseases include HHV6, EBV, Lyme disease and mycoplasma.
- Mercury exposure has been linked with an increased risk of developing multiple sclerosis, possibly due to mercury’s ability to destroy the protective myelin sheath leading to neuron death.
Symptoms of Multiple Sclerosis
- Visual disturbances
- Decrease or loss of balance and fine motor coordination
- Muscle weakness
- Sensations such as numbness, prickling, or “pins and needles” of the arms and legs
- Thinking and memory problems
- Constipation and urinary retention
- Constant and Chronic Fatigue worse after a hot shower or exercise
For answers to your questions or to make an appointment, call us toll-free TODAY at 800-923-7878 to speak with a Patient Care Representative. Let us help you get your life back.
Research on Multiple Sclerosis
Noseworthy, J. Progress in determining the causes and treatment of multiple sclerosis. Nature 399, A40–A47 (1999). https://doi.org/10.1038/399a040
The cause of multiple sclerosis remains unknown after more than a century of study. Unconfirmed work has once more indicated that a viral infection may be important in the aetiology of the disease, and there is considerable evidence for an important genetic influence on disease susceptibility. The clinical course is as variable as that of any disease in medicine. Studies using serial magnetic resonance imaging have helped to define the disease course and response to experimental therapies.
Although the predominant pathological characteristic is myelin loss with preservation of axons, some studies recall classic descriptions that irreversible axonal destruction may occur, perhaps even in the early stages of the illness. There are now several, partially effective therapies for relapsing forms of multiple sclerosis and here I review progress in determining the timing and course of the illness and the steps that need to be taken to identify more effective treatments for this disease.
Gilden DH. Infectious causes of multiple sclerosis. Lancet Neurol. 2005 Mar;4(3):195-202. doi: 10.1016/S1474-4422(05)01017-3. Erratum in: Lancet Neurol. 2005 May;4(5):269. PMID: 15721830; PMCID: PMC7129502.
Multiple sclerosis (MS) is a serious chronic neurological disorder in which demyelination and inflammation occur in the white matter of the CNS. The findings of many epidemiological studies and a discordance of MS in monozygotic twins suggest that the disorder is acquired. The most likely cause is a virus because more than 90% of patients with MS have high concentrations of IgG, manifest as oligoclonal bands, in the brain and CSF.
Most chronic inflammatory CNS disorders are infectious. More indirect evidence that MS is caused by a virus is the association of several viruses with demyelinating encephalomyelitis in human beings, and the induction of demyelination in animals infected with viruses in research. Nevertheless, no virus has been isolated from the brains of patients who had MS. Molecular analysis of IgG gene specificity in the brain and CSF of those with MS has shown features of an antigen-driven response: clonal amplification and extensive somatic mutations. A viral antigen against which the IgG in MS brain and CSF is directed might be identified.